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ΔNp63α is an oncogene that targets chromatin remodeler to

Mice, Phosphoproteins / genetics; Phosphoproteins / metabolism*; Polymerase Chain Reaction; Protein Binding; Skin / cytology*; Stem Cells

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Chromatin Remodeling Factor Drives Cancer Progression by

Subsequently, we confirmed the inverse regulation of FH and protein expression in transplanted tumors from mice (Supplementary Fig.

interacts with and stabilizes GINS4 transcript that promotes

mice injected with GINS4-expressing PC9 cells or control cells. n-p A xenograft mouse model was established, and the volume of tumors derived from

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Retinoic acid promotes ferroptosis by attenuating expression

mice after treatment with ATRA. (F, G) RT-qPCR analysis of ferroptosis-associated genes in ATRA-treated A549 cells in culture (F) or in ATRA-treated

USP11-mediated deubiquitination inhibits ferroptosis in

In all, 24 5-week-old female athymic BALB/c mice with comparable body weight were randomly assigned to 4 groups, with 6 mice per group.

The ubiquitin‐specific protease 5 mediated deubiquitination of

And tumor growth was suppressed in mice receiving USP5 knockout transplants (Figure 5K, Figure S4H). In order to further detect whether the